Description
A scientific and comprehensive approach to fat loss and body recomposition. The morphic programming in this targets bodyfat from many angles. Testers came back with positive feedback, some are ecstatic.
Stacks very well with Golden Era Physique, Morphic Test and Morphic GH.
THE SHREDDED EFFECT
Mitochondria Uncoupling:
Uncoupling mitochondria in all muscle groups to allow for a larger consumption of fat. Thereby making muscles appear more defined.
Fat Metabolism via PHD3 Inhibition:
The enzyme prolyl hydroxylase 3 (PHD3) is inhibited to metabolise fat. PHD3 basically inhibits unnecessary’ fat metabolism so we inhibit PHD3 production allowing for greater fat metabolism.
Thermogenesis via UCP genes:
Ucp1 and Uep2 (thermogenin proteins) are activated to produce heat and which in turn causes fat loss.
IRX3 & IRXS (Genes) deactivated:
These genes basically tell the body to store energy as fat, deactivate them and body fat will be utilised instead of food.
Chlorogenic Acid:
This causes your body to generate chlorogenic acid which inhibits fat and sugar absorption along with Lipolysis (which is the breakdown of lipids; it involves the hydrolysis of triglycerides into free fatty acids).
PPAR Delta(Cardarine effect):
It is a transcription factor that regulates energy metabolism in muscles and plays a key role in the adaptation of muscles to endurance exercise. Increase the expression of PPAR delta so it improves energy metabolism in muscles, leading to enhanced endurance performance.
PPAR-Alpha:
Activation of PPAR-u peroxisome (gene to produce protein) in muscles, heart, and liver areas and directed to receptors. Peroxisomes are compartments where cells turn fatty molecules into energy and useful materials, like the myelin sheaths that protect nerve cells. Peroxisome dysfunction has,been linked to severe metaboliΔ disorders, and peroxisomes may have wider significance for neurodegeneration, obesity, cancer, and age-related disorders.
AMPK Activation:
AMP-activated protein kinase (AMPK) is an enzyme that regulates cellular energy homeostasis. By activating AMPK, this will increase the body’s ability to generate energy from glucose and fat, leading to improved endurance and fat loss.
Fat Redirection:
Turns fat surrounding your muscles into a fuel source for your muscles.
Mimic 100 mg dose of 5 Amino 1MQ:
It is actually a tiny molecule that disrupts the function of the enzyme called NNMT. NNMT stands for nicotinamide N-methyltransferase. NNMT is actually an enzyme that acts to slow down fat cell metabolism (fat burning).
As fat cells grow, they overproduce NNMT enzyme. This slowdown makes it harder for these cells to burn accumulating fat. As fat tissue grows and more NNMT is produced, greater amounts of hormones and pro-inflammatory signals are produced that are responsible for weight gain.
5 Amino 1MQ disrupts the NNMT process, it can then increase cellular energy regulators, prevent lipogenesis (fat accumulation), treat related metabolic conditions, increase NAD+ and SAM concentrations in fat cells, reverse diet induced obesity and also regulate energy expenditure in fat cells.
Mimic 25 mg dose of MOTS-c:
An exercise-mimetic, meaning it imitates exercise on the body. Exercise also increases muscle glucose uptake without stimulating insulin.
Mimic 40 mg dose of Stenabolic (SR-9009):
SR-9009 -a highly potent performance-enhancing agent, commonly called “exercise in a bottle compound,” “automated workout pill,” or “metabolic modulator.” Studies revealed its unique potential to accelerate fat loss and muscle hypertrophy.
Stenabolic (SR-9009) is the agonist of REV-ERB nuclear receptors alpha and beta.
Beta 2 Agonist (Clen Effect):
Clenbuterol is a beta-2 andrenergic receptor agonist and has very strong stimulating and metabolism boosting properties.
Optially high T4 Thyroid Hormone Release with optimal conversion of t4 to t3 fofat loss.
Metformin:
Designed to safely simulate the insulin sensitivity enhancing effects of a sizeabldose of glocophage
Abdominals:
More specific fat apoptosis of subcutaneous fat tissue in the abdominal region.
Myostatin Mutation:
Recreate the C313Y mutation in myostatin directly and only in muscle cells. Directed activation of Myosatellite cells, also known as satellite cells or muscle stem cells, are small multipotent cells with very little cytoplasm found in mature muscle. Fat cells shall be converted to Muscle Mvosatellite cells or muscle stem cells.
A directed push ofnutrients toward muscle cells to help with their growth and development.
Rev-ErbA targeted to all muscle groups along with apoptosis of fat cells.
PDGF-AB:
Platelet derived growth factor AB targeted directly to muscles.
Increased mTOR activation for higher protein synthesis.
Increased pi3k expression in skeletal muscles.
7 to 10 min at a time
Start with twice a day
Don’t listen at night before sleep
